Remyelination strategies: New advancements toward a regenerative treatment in multiple sclerosis
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Summary
Despite the encouraging experimental findings and the constantly increasing knowledge of the molecular mechanisms of remyelination and remYelination failure, there remain many questions before this knowledge can be successfully translated into clinical trials.
- Type
- article
- Published
- 2006-05-01
- Cited by
- 16
- References
- 68
- OpenAlex
- https://openalex.org/W16635432
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:12497077
Keywords
Dome (geology), Geology, Geomorphology
References
- Functional Genomic Analysis of Remyelination Reveals Importance of Inflammation in Oligodendrocyte Regeneration
- Absence of Fibroblast Growth Factor 2 Promotes Oligodendroglial Repopulation of Demyelinated White Matter
- In vivo analysis of oligodendrocyte lineage development in postnatal FGF2 null mice
- The ErbB4 Neuregulin Receptor Mediates Suppression of Oligodendrocyte Maturation
- Minocycline-mediated inhibition of microglia activation impairs oligodendrocyte progenitor cell responses and remyelination in a non-immune model of demyelination.
- Combinatorial Profiles of Oligodendrocyte-Selective Classes of Transcriptional Regulators Differentially Modulate Myelin Basic Protein Gene Expression
- Human Monoclonal IgM Antibody Promotes CNS Myelin Repair Independent of Fc Function
- Remyelinating strategies for the treatment of multiple sclerosis.
- Inhibition of Notch signaling enhances tissue repair in an animal model of multiple sclerosis.
- Neurosphere-derived multipotent precursors promote neuroprotection by an immunomodulatory mechanism
- Expression of the chemokine receptors CXCR1 and CXCR2 in rat oligodendroglial cells.
- A pilot study of recombinant insulin-like growth factor-1 in seven multiple sclerosis patients
- Mechanisms of CNS remyelination--the key to therapeutic advances.
- Myelinogenesis and Axonal Recognition by Oligodendrocytes in Brain Are Uncoupled in Olig1-Null Mice
- Erythropoietin: Novel Approaches to Neuroprotection in Human Brain Disease
- Neuregulin-1 type III determines the ensheathment fate of axons.
- Protective autoimmunity and neuroprotection in inflammatory and noninflammatory neurodegenerative diseases.
- Immunoglobulins promote remyelination in the central nervous system
- Fibroblast growth factor 2 induces loss of adult oligodendrocytes and myelin in vivo.
- CXC chemokine receptors on human oligodendrocytes: implications for multiple sclerosis.
Cited by
- The chemokine receptor CXCR2 is differentially regulated on glial cells in vivo but is not required for successful remyelination after cuprizone‐induced demyelination
- Remyelination after cuprizone induced demyelination is accelerated in mice deficient in the polysialic acid synthesizing enzyme St8siaIV.
- The cuprizone animal model: new insights into an old story
- Magnetic nanoparticle-mediated gene transfer to oligodendrocyte precursor cell transplant populations is enhanced by magnetofection strategies.
- Nonmuscle myosin-2: mix and match
- Sequential myelin protein expression during remyelination reveals fast and efficient repair after central nervous system demyelination
- Magnetic nanoparticles for oligodendrocyte precursor cell transplantation therapies: progress and challenges
- New insights into an old story
- Applications of magnetic particles for oligodendrocyte precursor cell transplantation strategies
- Damage and Repair in Experimental Cortical Demyelination
- Mice lacking Gpr37 exhibit decreased expression of the myelin-associated glycoprotein MAG and increased susceptibility to demyelination
- Molecular basis of the axon-oligodendrocyte interaction during remyelination
- From Blurred Vision into Clarity - Unveiling the Diagnosis of Adolescent Onset Multiple Sclerosis
- Smith ScholarWorks Smith ScholarWorks
- Regional differences of molecular factors during demyelination and early remyelination in the CNS
- Différenciation dirigée de cellules précurseurs d'oligodendrocytes vers un destin cellulaire myélinisant
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