RA-RhoGAP, Rap-activated Rho GTPase-activating Protein Implicated in Neurite Outgrowth through Rho*♦
Explore this paper's citation graph
Summary
Results indicate that RA-RhoGAP transduces a signal from Rap1 to Rho and regulates the neurite outgrowth.
- Type
- article
- Published
- 2005-09-23
- Cited by
- 69
- References
- 47
- Access
- Open access
- OpenAlex
- https://openalex.org/W16014623
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:23462222
Keywords
Bureaucracy, Computer science, Political science, Law, Politics
References
- Bni1p implicated in cytoskeletal control is a putative target of Rho1p small GTP binding protein in Saccharomyces cerevisiae.
- Asparagine residue in the rho gene product is the modification site for botulinum ADP-ribosyltransferase.
- RHO GTPASES in neuronal morphogenesis
- Regulation of vascular endothelial barrier function by Epac, a cAMP-activated exchange factor for Rap GTPase.
- Neuronal polarity and trafficking.
- Nectin and afadin: novel organizers of intercellular junctions
- Involvement of the Annexin II-S100A10 Complex in the Formation of E-cadherin-based Adherens Junctions in Madin-Darby Canine Kidney Cells*
- Breaking the neuronal sphere: regulation of the actin cytoskeleton in neuritogenesis
- TAB2, a novel adaptor protein, mediates activation of TAK1 MAPKKK by linking TAK1 to TRAF6 in the IL-1 signal transduction pathway.
- Identification of RARhoGAP, a novel putative RhoGAP gene expressed in male germ cells.
- Mitogen-activated Protein Kinase Kinase Kinase Kinase 4 as a Putative Effector of Rap2 to Activate the c-Jun N-terminal Kinase*
- A critical role for a Rho-associated kinase, p160ROCK, in determining axon outgrowth in mammalian CNS neurons.
- A stimulatory GDP/GTP exchange protein for smg p21 is active on the post-translationally processed form of c-Ki-ras p21 and rhoA p21.
- Involvement of Rho and Rac small G proteins and Rho GDI in Ca2+‐dependent exocytosis from PC12 cells
- Cyclic AMP-independent Involvement of Rap1/B-Raf in the Angiotensin II AT2 Receptor Signaling Pathway in NG108-15 Cells*
- Rho GTPases in growth cone guidance.
- Hippocampal neuronal polarity specified by spatially localized mPar3/mPar6 and PI 3-kinase activity.
- Modulation of synapse formation by cyclic adenosine monophosphate.
- RAPL: taking the Rap in immunity
- Preferential addition of newly synthesized membrane protein at axonal growth cones
Cited by
- RhoA GTPase inhibition organizes contraction during epithelial morphogenesis
- Adquant; an image analysis toolbox for the automated, quantitative investigation of cell adhesion
- Biochemische Charakterisierung neuartiger GTPase aktivierender Proteine der Rho-Familie aus Pflanzen
- Imaging the translocations of CLIC4 and Epac1
- Sequential activation of Rap1 and Rac1 small G proteins by PDGF locally at leading edges of NIH3T3 cells
- Molecular switches in CFTR processing and trafficking: the role of the novel interactor Rap1A
- Signaling Pathways That Control Rho Kinase Activity Maintain the Embryonic Epicardial Progenitor State
- Rap1 signaling in endothelial barrier control
- Neurite outgrowth from PC12 cells by basic fibroblast growth factor (bFGF) is mediated by RhoA inactivation through p190RhoGAP and ARAP3
- Retracted: Ubiquitination of the GTPase Rap1B by the ubiquitin ligase Smurf2 is required for the establishment of neuronal polarity
- Small G proteins and their regulators in cellular signalling.
- Regulation of neurite outgrowth by G(i/o) signaling pathways.
- Cyclic AMP-Rap1A signaling activates RhoA to induce α(2c)-adrenoceptor translocation to the cell surface of microvascular smooth muscle cells.
- Current knowledge of the large RhoGAP family of proteins
- cAMP–Epac2‐mediated activation of Rap1 in developing male germ cells: RA‐RhoGAP as a possible direct down‐stream effector
- GEFs and GAPs: critical elements in the control of small G proteins.
- Role of the Go/i signaling network in the regulation of neurite outgrowth.
- Cell Migration: Regulation of cytoskeleton by Rap1 in Dictyostelium discoideum
- The cAMP-responsive Rap1 Guanine Nucleotide Exchange Factor, Epac, Induces Smooth Muscle Relaxation by Down-regulation of RhoA Activity*
- E-cadherin dis-engagement activates the Rap1 GTPase