Calcium channel antagonist receptors in cerebral cortex from alcoholic patients.
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Summary
No difference in (+)-[3H]PN 200-110 binding was observed, suggesting that ethanol-induced changes in Ca2+ channel expression are more likely to be related to acute withdrawal than to chronic ethanol exposure alone.
- Type
- article
- Published
- 1989-01-23
- Cited by
- 19
- References
- 16
- OpenAlex
- https://openalex.org/W2538202
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:33235663
Keywords
Computer science
References
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- The Role of Abstinence in the Genesis of Alcoholic Epilepsy
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- Inhibition of synaptosomal calcium uptake by ethanol.
Cited by
- Neuroplastic alterations in the limbic system following cocaine or alcohol exposure.
- Ethanol and the nervous system.
- Ethanol and neuronal calcium channels.
- Smithkline Beecham Prize for Young Psychopharmacologists
- Diltiazem alters some withdrawal signs in pentobarbital-dependent rats.
- Nifedipine alleviates alterations in hippocampal kindling after repeated ethanol withdrawal.
- Potentiation of ethanol via interference with calcium channels.
- Neuroadaptive responses to chronic ethanol.
- Pharmacotherapies for alcoholism: promising agents and clinical issues.
- N-methyl-D-aspartate glutamate receptors and alcoholism: reward, dependence, treatment, and vulnerability.
- Chronic ethanol treatment alters omega-conotoxin and Bay K 8644 sensitive calcium channels in rat striatal synaptosomes.
- Chronic ingestion of ethanol increases the number of Ca2+ channels of hippocampal neurons of long-sleep but not short-sleep mice.
- Nimodipine in acute alcohol withdrawal state.
- Current concepts of ethanol dependence.
- Alcohol, nitric oxide, and neurotoxicity: is there a connection?--a review.
- Pharmacotherapy and Pathophysiology of Alcohol Withdrawal
- Effects of Ethanol on Voltage-Dependent Calcium Channel Function
- Research Advances in Pharmacotherapy for Alcoholism
- Suchttherapeutika zur Sucht
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