Kappa B-type enhancers are involved in lipopolysaccharide-mediated transcriptional activation of the tumor necrosis factor alpha gene in primary macrophages
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Summary
It is speculated that NF-kappa B and/or related proteins are involved in the LPS-induced transcriptional activation of the TNF-alpha gene, and that factors interacting with the Y box can additionally modulate the activity of the gene in macrophages.
- Type
- article
- Published
- 1990-01-01
- Cited by
- 848
- References
- 39
- Access
- Open access
- OpenAlex
- https://openalex.org/W2104921
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:12942446
Keywords
Performance measurement, Team composition, Team effectiveness, Computer science, Performance appraisal
References
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- Activation of HIV gene expression during monocyte differentiation by induction of NF-kB
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- B-cell-specific enhancer activity of conserved upstream elements of the class II major histocompatibility complex DQB gene.
- Nuclear proteins interacting with the promoter region of the human granulocyte/macrophage colony-stimulating factor gene.
- Molecular control of tissue‐specific expression at the mouse TNF locus
- Production of tumor necrosis factor/cachectin by human B cell lines and tonsillar B cells
Cited by
- Ionizing radiation induces expression and binding activity of the nuclear factor kappa B.
- In situ changes in the relative abundance of human epidermal cytokine messenger RNA levels following exposure to the poison ivy/oak contact allergen urushiol
- Global cerebral ischemia activates nuclear factor-kappa B prior to evidence of DNA fragmentation.
- Human Tumor Necrosis Factor-α Gene 3′ Untranslated Region Confers Inducible Toxin Responsiveness to Homologous Promoter in Monocytic THP-1 Cells*
- p38 map kinase regulates TNF-alpha production in human astrocytes and microglia by multiple mechanisms.
- Tumor Necrosis Factor and Lymphotoxin: Molecular Genetics, Regulation and Physiological Role
- Increased Adhesion Molecule Expression in Serosal Fibroblasts Isolated From Patients with Inflammatory Bowel Disease is Secondary to Inflammation
- The role of p105 protein in NFκB activation in ANA‐1 murine macrophages following stimulation with titanium particles
- Liposomal Delivery of Heat Shock Protein 72 Into Renal Tubular Cells Blocks Nuclear Factor-&kgr;B Activation, Tumor Necrosis Factor-&agr; Production, and Subsequent Ischemia-Induced Apoptosis
- Pneumocystis Colonization and Chronic Obstructive Pulmonary Disease in a Simian Model of HIV Infection
- Inhibition of nuclear factor-kappa B prevents staphylococcal enterotoxin A-induced fever
- Rhinovirus Replication in Human Macrophages Induces NF-κB-Dependent Tumor Necrosis Factor Alpha Production
- Glycyrrhiza inflata-derived chalcones, Licochalcone A, Licochalcone B and Licochalcone D, inhibit phosphorylation of NF-kappaB p65 in LPS signaling pathway.
- Effect of protein-energy malnutrition on nuclear factor kappa B activation following global ischemia
- Cardiac Fibroblast GRK2 Deletion Enhances Contractility and Remodeling Following Ischemia/Reperfusion Injury
- Lipopolysaccharide Signals Activation of Tumor Necrosis Factor Biosynthesis Through the Ras/Raf-1/MEK/MAPK Pathway
- Innate immune responses in the lung and liver
- Toxic shock syndrome toxin-1, toxic shock, and the immune system.
- In situ detection of stage-specific genes and enhancers in B cell differentiation via gene-search retroviruses.
- The Role of MKK3 in Mediating Signals to the p38 MAP Kinase Pathway: A Dissertation
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