Evidence for the effects of a superantigen in rheumatoid arthritis.
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- Type
- article
- Published
- 1991-07-19
- Cited by
- 638
- References
- 39
- OpenAlex
- https://openalex.org/W1857971
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:11214278
Keywords
Political science
References
- Linkage of Mls genes to endogenous mammary tumour viruses of inbred mice
- An endogenous retrovirus mediating deletion of αβ T cells?
- HLA-DR genotype risks in seropositive rheumatoid arthritis.
- Synovial fluid lymphocytes differ from peripheral blood lymphocytes in patients with rheumatoid arthritis.
- Extrathymic tolerance of mature T cells: clonal elimination as a consequence of immunity.
- Selective anergy of V beta 8+,CD4+ T cells in Staphylococcus enterotoxin B-primed mice
- Ia+ T cells in synovial fluid and tissues of patients with rheumatoid arthritis.
- Treatment of intractable rheumatoid arthritis with total lymphoid irradiation.
- Lymphocyte depletion by continuous flow cell centrifugation in rheumatoid arthritis: clinical effects.
- T cell tolerance by clonal elimination in the thymus.
- T cells can distinguish between allogeneic major histocompatibility complex products on different cell types
- The shared epitope hypothesis. An approach to understanding the molecular genetics of susceptibility to rheumatoid arthritis.
- A nondeletional mechanism of thymic self tolerance.
- The T-cell repertoire is heavily influenced by tolerance to polymorphic self-antigens
- Tolerance in T-cell-receptor transgenic mice involves deletion of nonmature CD4+8+ thymocytes
- The V beta-specific superantigen staphylococcal enterotoxin B: stimulation of mature T cells and clonal deletion in neonatal mice.
- Selective expansion of T cells expressing V beta 2 in toxic shock syndrome
- Self-tolerance eliminates T cells specific for Mls-modified products of the major histocompatibility complex
- Programmed cell death and extrathymic reduction of Vβ8+CD4+ T cells in mice tolerant to Staphylococcus aureus enterotoxin B
- In vivo induction of anergy in peripheral V beta 8+ T cells by staphylococcal enterotoxin B
Cited by
- An experimentally validated panel of subfamily‐specific oligonucleotide primers (Vα1‐w29/Vβ1‐w24) for the study of human T cell receptor variable V gene segment usage by polymerase chain reaction
- The use of the PCR to quantitate gene expression.
- Evidence for an Endogenous Superantigen Deleting Human Vβ2 Positive T‐Lymphocytes
- Superantigen-reactive human T cells express a biased repertoire of T-cell receptor V beta joining regions.
- Correlation between infection and the onset of the giant cell (temporal) arteritis syndrome. A trigger mechanism?
- T cell influence on superantigen-induced arthritis in MRL-lpr/lpr mice.
- Influence of non-inherited maternal HLA on disease development
- Mycoplasma arthritidis-derived superantigen induces proinflammatory monokine gene expression in the THP-1 human monocytic cell line
- The role of T cells in the immunopathogenesis of rheumatoid arthritis: new perspectives.
- Reactivity Patterns of Synovial T‐Cell Lines Derived from a Patient with Rheumatoid Arthritis. I. Reactions with Defined Antigens and Auto‐Antigens Suggest the Existence of Multireactive T‐Cell Clones
- Idiopathic dilated cardiomyopathy: a superantigen-driven autoimmune disease.
- The arthritogenic properties of microbial antigens. Their implications in disease states.
- Superantigens and their role in autoimmune disorders.
- Immunotherapeutic strategies directed at the trimolecular complex.
- Intestinal inflammation and the gut microflora.
- Detection of bacterial superantigens using HLA characterisation of the TCR class II and V signature
- Interferon-gamma-producing T cells in giant cell vasculitis represent a minority of tissue-infiltrating cells and are located distant from the site of pathology.
- Characterization of human T cell clones specific for coronavirus 229E.
- Superantigens and their potential role in human disease.
- Superantigen-mediated lethal shock: the functional state of ligand-reactive T cells.
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