Experimental Evidence of Ischemic Thresholds and Functional Recovery
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Summary
Reestablishment of sufficient perfusion must be induced very early after an ischemic attack to ameliorate the potentially harmful biochemical sequelae of transient ischemia.
- Type
- article
- Published
- 1992-11-01
- Cited by
- 161
- References
- 41
- Access
- Open access
- OpenAlex
- https://openalex.org/W1440719
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:9979645
Keywords
Library instruction, Computer science, Discovery learning, Bibliography, World Wide Web
References
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- Calcium Fluxes, Calcium Antagonists, and Calcium-Related Pathology in Brain Ischemia, Hypoglycemia, and Spreading Depression: A Unifying Hypothesis
- Excitatory amino acid receptors and ischemic brain damage in the rat.
- Relationship between the cortical evoked potential and local cortical blood flow following acute middle cerebral artery occlusion in the baboon.
- Differences in ischemia-induced accumulation of amino acids in the cat cortex.
- Recovery of integrative central nervous function after one hour global cerebro-circulatory arrest in normothermic cat.
- Excitotoxity and the NMDA receptor
- Therapeutic window of halothane anesthesia for reversal of delayed neuronal injury in gerbils: relationship to postischemic motor hyperactivity.
- Delayed neuronal death in the gerbil hippocampus following ischemia.
- Calcium, neuronal hyperexcitability and ischemic injury.
Cited by
- Ischaemic brain tissue salvaged from infarction with alteplase.
- Localizing Acute Stroke-related EEG Changes:: Assessing the Effects of Spatial Undersampling
- The ischemic penumbra: how does tissue injury evolve?
- Microglia as a unique cellular target in the treatment of stroke: potential neurotoxic mediators produced by activated microglia.
- The Ischemic Penumbra: Correlates in Imaging and Implications for Treatment of Ischemic Stroke
- CRITICAL ROLE OF EUKARYOTIC TRANSLATION INITIATION FACTOR 4G IN MEDIATING ISCHEMIA-INDUCED NEURONAL DEATH
- Decompressive hemicraniectomy in a new cat model. Methodological description of the PET study protocol.
- Original Communication Beneficial vascular and metabolic effects of cobalt-ATP in spontaneously hypertensive rabbits with diffuse chronic cerebral ischaemia
- Imaging the pathophysiology of ischemic cerebrovascular disease
- Mechanisms of motor dysfunction after transient MCA occlusion: persistent transmission failure in cortical synapses is a major determinant.
- Angiotensin-II-Rezeptorantagonisten und endotheliale Progenitorzellen: ein neuer therapeutischer Ansatz?
- Application of proteomics to cerebrovascular disease
- Reperfusion injury after stroke: neurovascular proteases and the blood-brain barrier.
- Cortical and subcortical contributions to ideomotor apraxia: analysis of task demands and error types.
- Dynamic CT perfusion to assess the effect of carotid revascularization in chronic cerebral ischemia.
- Evaluation the development of focal cerebral ischemia in rats by optical imaging based on the spreading depression signals
- The therapeutic window for spinal cord decompression in a rat spinal cord injury model.
- Alterations of local cerebral blood flow, phorbol 12,13-dibutyrate binding activity, and histological damage during acute focal ischaemia in rat brain
- The history of diagnosing carotid sinus hypersensitivity: why are the current criteria too sensitive?
- Amino acid uptake in ischemically compromised brain tissue.
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