The dynamic genetic architecture of early childhood BMI
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Summary
This work characterizes the dynamic genetic architecture of childhood BMI during the first 8 years of life by identifying genetic loci involved in the leptin–melanocortin pathway.
- Type
- letter
- Published
- 2022-03-01
- Cited by
- 0
- References
- 12
- Access
- Open access
- OpenAlex
- https://openalex.org/W4220800656
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:247598811
Keywords
Genetic architecture, Architecture, Childhood obesity, Obesity, Melanocortin
References
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- The Association of Adolescent Obesity with Risk of Severe Obesity in Adulthood
- An Overview of Mongenic and Syndromic Obesities in Humans
- Meta-analysis of genome-wide association studies for height and body mass index in ∼700,000 individuals of European ancestry
- Clinical, molecular genetics and therapeutic aspects of syndromic obesity
- Maternal and fetal genetic effects on birth weight and their relevance to cardio-metabolic risk factors
- Glucagon, GLP-1 and Thermogenesis
- GWAS on longitudinal growth traits reveals different genetic factors influencing infant, child, and adult BMI
- Genome-wide association study reveals dynamic role of genetic variation in infant and early childhood growth
- The melanocortin pathway and energy homeostasis: From discovery to obesity therapy
- Characterization of the genetic architecture of infant and early childhood body mass index
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