Leucine-rich repeat and sterile alpha motif containing 1 promotes the oncogenic growth of human hepatocellular carcinoma cells
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Summary
The data suggest that LRSAM1 promotes the oncogenic growth of human HCC cells, although the underlying mechanisms remain to be explored.
- Type
- article
- Published
- 2019-10-03
- Cited by
- 4
- References
- 37
- Access
- Open access
- OpenAlex
- https://openalex.org/W2977919598
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:203638864
Keywords
Gene knockdown, Ectopic expression, Cancer research, Hepatocellular carcinoma, Ubiquitin ligase
References
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- Autophagy fights disease through cellular self-digestion
- The contributions of hepatitis B virus and hepatitis C virus infections to cirrhosis and primary liver cancer worldwide.
- The LRR and RING Domain Protein LRSAM1 Is an E3 Ligase Crucial for Ubiquitin-Dependent Autophagy of Intracellular Salmonella Typhimurium
- Hepatocellular cancer: a guide for the internist.
- Autophagy and cancer cell metabolism
- Autophagy and Metabolism
- LRSAM1, an E3 Ubiquitin Ligase with a Sense for Bacteria
- Autophagy in mammalian development and differentiation
- Cullins and Cancer
- Tumor suppression by autophagy through the management of metabolic stress.
- The expression of leucine-rich repeat gene family members in colorectal cancer
- Persistent activation of Nrf2 through p62 in hepatocellular carcinoma cells
- Tal, a Tsg101-specific E3 ubiquitin ligase, regulates receptor endocytosis and retrovirus budding.
- Global cancer statistics
- Annual Report to the Nation on the Status of Cancer, 1975–2011, Featuring Incidence of Breast Cancer Subtypes by Race/Ethnicity, Poverty, and State
Cited by
- Synthesis, Antimicrobial Potency with In Silico Study of Boc-Leucine-1,2,3-Triazoles.
- LRSAM1 mediated the degradation of intracellular Vibrio through the ubiquitination-autophagy-lysosome pathway in oyster
- Cannabidiol inhibits melanoma progression by regulating PPARγ-TET1 complex-dependent LRSAM1 demethylation.
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