The "Red Queen" Is Up and Running: Virus-Host Interactions Revealed Through the Prism of Multiomics.
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- Type
- letter
- Published
- 2019-08-01
- Cited by
- 2
- References
- 21
- Access
- Open access
- OpenAlex
- https://openalex.org/W2949563374
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:199593582
Keywords
Queen (butterfly), Computational biology, Biology, Zoology
References
- The contributions of hepatitis B virus and hepatitis C virus infections to cirrhosis and primary liver cancer worldwide.
- Innate and adaptive immune responses in HCV infections.
- Prevalence of steatosis and insulin resistance in patients with chronic hepatitis B compared with chronic hepatitis C and non-alcoholic fatty liver disease.
- Hepatitis C Virus, Cholesterol and Lipoproteins — Impact for the Viral Life Cycle and Pathogenesis of Liver Disease
- A new evolutionary law
- Activation of STAT3 by the Hepatitis C Virus Core Protein Leads to Cellular Transformation
- Diminished viral replication and compartmentalization of hepatitis C virus in hepatocellular carcinoma tissue
- Molecular mechanisms of hepatitis C virus-induced hepatocellular carcinoma.
- Risk of cirrhosis-related complications in patients with advanced fibrosis following hepatitis C virus eradication.
- Using Big Data to Discover Diagnostics and Therapeutics for Gastrointestinal and Liver Diseases
- Global prevalence and genotype distribution of hepatitis C virus infection in 2015: a modelling study.
- Transcriptional response to hepatitis C virus infection and interferon‐alpha treatment in the human liver
- Why highly effective drugs are not enough: the need for an affordable solution to eliminating HCV
- Hepatitis B Virus Does Not Interfere With Innate Immune Responses in the Human Liver.
- Mechanisms of NAFLD development and therapeutic strategies
- Oncogenic Signaling Induced by HCV Infection
- Dysregulation of the cohesin subunit RAD21 by Hepatitis C virus mediates host–virus interactions
- HCV-Induced Epigenetic Changes Associated With Liver Cancer Risk Persist After Sustained Virologic Response
- Combined Analysis of Metabolome, Proteomes, and Transcriptomes of HCV-infected Cells and Liver to Identify Pathways Associated With Disease Development
- Hepatitis C virus leaves an epigenetic signature post cure of infection by direct-acting antivirals
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