Severe oxidative stress in an acute inflammatory demyelinating model in the rhesus monkey
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Summary
Results indicate that pathogenic mechanisms in the rhesus EAE model may differ from the marmoset EAE and MS brain due to the neutrophil involvement, but may in the end lead to similar induction of oxidative stress and injury.
- Type
- article
- Published
- 2017-11-14
- Cited by
- 12
- References
- 40
- Access
- Open access
- OpenAlex
- https://openalex.org/W2768326041
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:28474487
Keywords
Marmoset, Multiple sclerosis, Experimental autoimmune encephalomyelitis, Oxidative stress, Encephalomyelitis
References
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Cited by
- Experimental autoimmune encephalomyelitis in the common marmoset: a translationally relevant model for the cause and course of multiple sclerosis
- Nrf2 Mediates the Anti-apoptotic and Anti-inflammatory Effects Induced by Gastrodin in Hydrogen Peroxide–Treated SH-SY5Y Cells
- Experimental Models of Neuroimmunological Disorders: A Review
- Rg3-enriched Korean Red Ginseng extract inhibits blood-brain barrier disruption in an animal model of multiple sclerosis by modulating expression of NADPH oxidase 2 and 4
- ADAM10 suppresses demyelination and reduces seizure susceptibility in cuprizone-induced demyelination model.
- Effects of biological sex and pregnancy in experimental autoimmune encephalomyelitis: It’s complicated
- Ebselen alleviates white matter lesions and improves cognitive deficits by attenuating oxidative stress via Keap1/Nrf2 pathway in chronic cerebral hypoperfusion mice.
- Ferroptosis and central nervous system demyelinating diseases
- Metal-induced autoimmunity in neurological disorders: A review of current understanding and future directions.
- Role of autoreactive Tc17 cells in the pathogenesis of experimental autoimmune encephalomyelitis
- Single-Cell RNA Sequencing Reveals Transcriptional Landscape of Neutrophils and Highlights the Role of TREM-1 in EAE
- Critical analysis of translational potential of rodent models of white matter pathology across a wide spectrum of human diseases
- Role of Autoreactive Tc17 Cells in the Pathogenesis of Experimental Autoimmune Encephalomyelitis (EAE)
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