Carvedilol exacerbate gentamicin-induced kidney mitochondrial alterations in adult rat.
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Summary
C carvedilol may contribute to the exacerbation of renal dysfunction induced by gentamicin, at least in some physiological and biochemical parameters.
- Type
- article
- Published
- 2017-02-01
- Cited by
- 11
- References
- 67
- OpenAlex
- https://openalex.org/W2550662381
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:19094010
Keywords
Gentamicin, Nephrotoxicity, Aminoglycoside, Carvedilol, Renal function
References
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- Membranes of mitochondria and chloroplasts
- Antibiotic use and abuse: A threat to mitochondria and chloroplasts with impact on research, health, and environment
- Protective effects of oral arabic gum administration on gentamicin-induced nephrotoxicity in rats.
- Carvedilol protects against the renal mitochondrial toxicity induced by cisplatin in rats.
- Cardiolipin Remodeling by ALCAT1 Links Oxidative Stress and Mitochondrial Dysfunction to Obesity
- Multiple pathways of cytochrome c release from mitochondria in apoptosis.
- REACTIVE OXYGEN SPECIES, CELLULAR REDOX SYSTEMS AND APOPTOSIS
- Metabolomic approaches to mitochondrial disease: correlation of urine organic acids.
- New insights into the mechanism of aminoglycoside nephrotoxicity: an integrative point of view.
- Mitochondria as ATP consumers in cellular pathology.
- Metformin prevents experimental gentamicin-induced nephropathy by a mitochondria-dependent pathway.
- Carvedilol: a beta blocker with antioxidant property protects against gentamicin-induced nephrotoxicity in rats.
- Role of reactive oxygen species and cardiolipin in the release of cytochrome c from mitochondria
- Aminoglycoside-Induced Nephrotoxicity
- Oxidative lipidomics of gamma-irradiation-induced intestinal injury.
Cited by
- Drug-Induced Mitochondrial Toxicity in the Geriatric Population: Challenges and Future Directions
- Ameliorative effect of gossypin against gentamicin‐induced nephrotoxicity in rats
- Protective Effects of Hydroalcoholic Extract of Nasturtium Officinale against Gentamicin Induced Mitochondrial Dysfunction in Rat Kidney
- Amelioration of renal and hepatic function, oxidative stress, inflammation and histopathologic damages by Malva sylvestris extract in gentamicin induced renal toxicity.
- Gentamicin Affects the Bioenergetics of Isolated Mitochondria and Collapses the Mitochondrial Membrane Potential in Cochlear Sensory Hair Cells
- Ursodeoxycholic acid abrogates gentamicin-induced hepatotoxicity in rats: Role of NF-κB-p65/TNF-α, Bax/Bcl-xl/Caspase-3, and eNOS/iNOS pathways.
- Cholesterol-functionalized carvedilol-loaded PLGA nanoparticles: anti-inflammatory, antioxidant, and antitumor effects
- Oxymatrine Reverses Gentamicin-Induced Nephrotoxicity via Ameliorating the Immune Response for Gentamicin Therapy
- Oxymatrine Alleviates Gentamicin-Induced Renal Injury in Rats
- The Impact of Micro-Nanoplastics on Mitochondria in the Context of Diet and Diet-Related Diseases
- Targeting the powerhouse: the mitochondrial perspective on gentamicin-induced kidney injury
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