Role of endothelial progenitor cells in cerebral aneurysm pathogenesis
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Summary
The results of the current study suggest that EPC dysfunction may contribute to CA formation, however, there is a lack of mechanistic data defining the molecular pathways by which EPC function is abrogated in patients with unruptured CAs.
- Type
- letter
- Published
- 2014-02-18
- Cited by
- 0
- References
- 5
- OpenAlex
- https://openalex.org/W2138492928
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:20447916
Keywords
Pathogenesis, Neurosurgery, Neuroradiology, Neurology, Progenitor cell
References
- Vascular Smooth Muscle Cells in Cerebral Aneurysm Pathogenesis
- Stromal Cell-Derived Factor-1 Is Associated with Angiogenesis and Inflammatory Cell Infiltration in Aneurysm Walls
- Decreased levels and function of circulating endothelial progenitor cells in unruptured intracranial saccular aneurysm patients
- EndMT contributes to the onset and progression of cerebral cavernous malformations
- Tumor Necrosis Factor-α Modulates Cerebral Aneurysm Formation and Rupture
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