Preferential Fas-mediated apoptotic execution at G1 phase: the resistance of mitotic cells to the cell death
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Summary
It is suggested that apoptotic execution is dependent on cell cycle phase and Fas-mediated apoptosis preferentially occurs at G1 phase.
- Type
- article
- Published
- 2012-05-01
- Cited by
- 8
- References
- 43
- Access
- Open access
- OpenAlex
- https://openalex.org/W2081845499
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:16919944
Keywords
Apoptosis, Cell biology, Cell cycle, Mitosis, Cytokinesis
References
- Phosphorylation of FADD/ MORT1 at Serine 194 and Association with a 70-kDa Cell Cycle-Regulated Protein Kinase1
- Specific resistance of T cells to CD95-induced apoptosis during S phase of the cell cycle.
- CD95‐mediated apoptosis: no variation in cellular sensitivity during cell cycle progression
- Cytotoxicity‐dependent APO‐1 (Fas/CD95)‐associated proteins form a death‐inducing signaling complex (DISC) with the receptor.
- Evidence that Fas-induced apoptosis leads to S phase arrest.
- Etoposide, topoisomerase II and cancer.
- Mammalian caspases: structure, activation, substrates, and functions during apoptosis.
- A Novel Protein That Interacts with the Death Domain of Fas/APO1 Contains a Sequence Motif Related to the Death Domain (*)
- Two CD95 (APO‐1/Fas) signaling pathways
- ZSTK474, a specific phosphatidylinositol 3-kinase inhibitor, induces G1 arrest of the cell cycle in vivo.
- FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis.
- Phosphorylation of FADD at serine 194 by CKIalpha regulates its nonapoptotic activities.
- Visualizing spatiotemporal dynamics of multicellular cell-cycle progression.
- Finishing mitosis, one step at a time
- Apoptosis by death factor.
- Timing of apoptosis onset depends on cell cycle progression in peripheral blood lymphocytes and lymphocytic leukemia cells.
- Fas-associated death domain protein interacts with methyl-CpG binding domain protein 4: A potential link between genome surveillance and apoptosis
- Susceptibility of rheumatoid arthritis synovial fibroblasts to FasL- and TRAIL-induced apoptosis is cell cycle-dependent
- Cooperative phosphorylation of FADD by Aur-A and Plk1 in response to taxol triggers both apoptotic and necrotic cell death.
- Mitosis: a matter of getting rid of the right protein at the right time.
Cited by
- Apoptosis induction-related cytosolic calcium responses revealed by the dual FRET imaging of calcium signals and caspase-3 activation in a single cell.
- Evolutionary analyses of caspase-8 and its paralogs: Deep origins of the apoptotic signaling pathways.
- The molecular mechanism of apoptosis upon caspase-8 activation: quantitative experimental validation of a mathematical model.
- Cyclin I is involved in the regulation of cell cycle progression
- Characterization of the anti-tumor activity of plants extracts from North Africa: Morocco
- Dehydrozaluzanin C inhibits colon cancer cell proliferation, apoptosis and cycle arrest through peroxisome proliferator-activated receptor γ (PPARγ) activation
- Fas signaling induces epithelial-mesenchymal transition through the ERK1/2 signaling pathway in colon cancer cells
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