Notch signalling is off and is uncoupled from HES1 expression in Ewing's sarcoma

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Summary

It is indicated that HES1 is uncoupled from Notch in ESFT, that EWS–FLI1‐mediated inhibition of Notch contributes to ESFT aggressive cell growth, and support a role for NotchIn ESFT tumour suppression, at least partly through the Notch effector HEY1.

Type
article
Published
2011-11-01
Cited by
34
References
60

Keywords

HES1, Notch signaling pathway, Biology, Cancer research, Notch proteins

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