Calcineurin-mediated LTD of GABAergic inhibition underlies the increased excitability of CA1 neurons associated with LTP.

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Summary

The tetanus that elicits NMDA-dependent LTP mediates a coordinately regulated double function that produces LTP of the EPSP and, concomitantly, LTD of the IPSP that leads to enhancement of E-S coupling.

Type
article
Published
2000-04-01
Cited by
305
References
42
Access
Open access

Keywords

Long-term potentiation, Neuroscience, Calcineurin, GABAergic, Chemistry

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