Clinical manifestations and erythrocyte adhesion to endothelium in sickle cell syndrome
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Summary
The possibility that a limited population of red cells could be responsible for the increase in red cell adhesion is investigated and the HbS concentration was found to be higher in the last washes compared to the first washes and could be partly explained by the presence of [3H] leucine‐labelled red cells in the dense fractions of sickle red cells separated by stractan gradient.
- Type
- article
- Published
- 1985-06-01
- Cited by
- 38
- References
- 15
- OpenAlex
- https://openalex.org/W2048987429
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:43031251
Keywords
Adhesion, Red Cell, Endothelium, Sickle cell anemia, Hemoglobinopathy
References
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- Hemostatic alterations accompanying sickle cell pain crises.
- Modulation of erythrocyte-endothelial interactions and the vasocclusive severity of sickling disorders.
- Adhesion of normal and sickle erythrocytes to endothelial monolayer cultures.
- Protein Synthesis in Reticulocytes Maturing IN VIVO
- An update on electrophoretic and chromatographic methods in the diagnosis of hemoglobinopathies.
- Erythrocyte adherence to endothelium in sickle-cell anemia. A possible determinant of disease severity.
- STUDIES OF THE RETICULOENDOTHELIAL MASS AND SEQUESTERING FUNCTION OF RAT BONE MARROW.
- Culture of human endothelial cells derived from umbilical veins. Identification by morphologic and immunologic criteria.
- Abnormal adherence of sickle erythrocytes to cultured vascular endothelium: possible mechanism for microvascular occlusion in sickle cell disease.
- Sickle Cell Crises: Ward Burdick Award Contribution
- Increased adhesion of erythrocytes to endothelial cells in diabetes mellitus and its relation to vascular complications.
- Separation of erythrocytes according to age on a simplified density gradient.
- Fibrinogen, a modulator of erythrocyte adhesion to vascular endothelium.
Cited by
- Decreased prostacyclin levels in sickle-cell disease.
- The Gárdos channel: a review of the Ca2+-activated K+ channel in human erythrocytes.
- Molecular basis of erythrocyte adhesion to endothelial cells in diseases
- Beyond hemoglobin polymerization: the red blood cell membrane and sickle disease pathophysiology.
- Priapism as a complication of sickle cell disease.
- The management of pain in sickle cell disease.
- Histamine as a Potential Initiator of Sickle Pain crisis by Mediation of Sickle Erythrocyte Adherence in a Shear-Dependent Manner
- Blood cell—Vessel wall interactions
- Rheologic predictors of the severity of the painful sickle cell crisis.
- Band 3 peptides block the adherence of sickle cells to endothelial cells in vitro.
- Advanced glycation end products, oxidant stress and vascular lesions
- Modification of membrane properties of erythrocytes by PGI2.
- Phenylhydrazine as a partial model for β‐thalassaemia red blood cell hemodynamic properties
- Endotoxin promotes adhesion of human erythrocytes to human vascular endothelial cells under conditions of flow
- Enhancement of sickle erythrocyte adherence to endothelium by autologous platelets
- Testosterone Induced Priapism in Two Adolescents with Sickle Cell Disease
- B‐CAM/LU expression and the role of B‐CAM/LU activation in binding of low‐ and high‐density red cells to laminin in sickle cell disease
- Sickle erythrocytes adhere to fibronectin-thrombospondin-integrin complexes exposed by thrombin-induced endothelial cell contraction.
- Unusually large von willebrand factor multimers preferentially promote young sickle and nonsickle erythrocyte adhesion to endothelial cells
- Activation State of α4β1 Integrin on Sickle Red Blood Cells Is Linked to the Duffy Antigen Receptor for Chemokines (DARC) Expression*
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