Behavioural and biochemical effects in C57BL/6J mice after a prolonged treatment with the δ‐opiate antagonist ICI 154129
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Summary
The data suggest that the locomotor response to morphine in C57 mice is not mediated through δ‐opiate receptors, and naloxone‐pretreated mice with respect to control are suggested.
- Type
- article
- Published
- 1984-12-01
- Cited by
- 3
- References
- 11
- Access
- Open access
- OpenAlex
- https://openalex.org/W2039496949
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:37400096
Keywords
Opiate, (+)-Naloxone, Antagonist, Morphine, Pharmacology
References
- Differential sensitivity to morphine-induced analgesia and motor activity in two inbred strains of mice: behavioral and biochemical correlations.
- Naltrexone-induced opiate receptor supersensitivity.
- Selective antagonists at the opiate delta-receptor.
- THE ATTRACTIONS OF PROTEINS FOR SMALL MOLECULES AND IONS
- Chronic naloxone results in prolonged increases in opiate binding sites in brain.
- Effects of morphine and chlordiazepoxide on avoidance behaviour in two inbred strains of mice.
- Difference in the development of tolerance to morphine and D-ALA2-methionine-enkephalin in C57 BL/6J and DBA/2J mice.
- Effects of morphine and chlordiazepoxide on locomotor activity in two inbred strains of mice.
- Chronic naltrexone increases opiate binding in brain and produces supersensitivity to morphine in the locus coeruleus of the rat.
Cited by
- Evidence from behavioural and in vitro receptor binding studies that the enkephalinergic system does not mediate acute ethanol effects.
- Unequal opiate cross-tolerance to morphine in the locomotor-activation model in the mouse.
- Brain μ and δ opioid receptors mediate different locomotor hyperactivity responses of the C57BL/6J mouse
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