Quantitative electron microscopic analysis of the penetration of VSV into L cells.
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Summary
There was a quantitative relationship between the disappearance of extracellular attached virus and the appearance of virus in invaginations, and, somewhat later, in small intracellular vacuoles.
- Type
- article
- Published
- 1974-03-01
- Cited by
- 38
- References
- 14
- OpenAlex
- https://openalex.org/W2011989881
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:43276320
Keywords
Penetration (warfare), Vesicular stomatitis virus, Virus, Centrifugation, Vacuole
References
- Entry of Vesicular Stomatitis Virus into L Cells
- Fusion of Vesicular Stomatitis Virus with the Cytoplasmic Membrane of L Cells
- Structure and Development of Viruses as Observed in the Electron Microscope IX. Entry of Parainfluenza I (Sendai) Virus
- Electron Microscopy of Herpes Simplex Virus
- Structure and development of viruses as observed in the electron microscope. 8. Entry of influenza virus.
- Electron microscopic observations on the nature of vaccinia virus particle aggregation.
- Early events in cell-animal virus interactions.
- Correlation between tumor invasiveness and content of A-type particles in Ehrlich ascites tumor: a model of quantitative electron microscopy.
- Viropexis of herpes simplex virus by HeLa cells.
- Rapid Adsorption of Vaccinia Virus on Tissue Culture Cells by Centrifugal Force.∗
- Viropexis of vesicular stomatitis virus by L cells.
- Attachment and penetration of influenza virus.
- Viropexis, the Mechanism of Influenza Virus Infection
Cited by
- Cell fusion by Semliki Forest, influenza, and vesicular stomatitis viruses
- Biologically active peptides of the vesicular stomatitis virus glycoprotein
- Inhibition of vesicular stomatitis virus infection by spike glycoprotein. Evidence for an intracellular, G protein-requiring step
- The entry into host cells of Sindbis virus, vesicular stomatitis virus and Sendai virus.
- Vesicular Stomatitis Virus Enters Cells through Vesicles Incompletely Coated with Clathrin That Depend upon Actin for Internalization
- Binding and entry of animal viruses
- Pathway of vesicular stomatitis virus entry leading to infection.
- Penetration of influenza examined by means of virus aggregates.
- Host Cell Factors and Functions Involved in Vesicular Stomatitis Virus Entry
- Monensin blocks endocytosis of vesicular stomatitis virus.
- Neutralized vesicular stomatitis virus binds to host cells by a different "receptor".
- Entry pathway of vesicular stomatitis virus into different host cells.
- A cell line expressing vesicular stomatitis virus glycoprotein fuses at low pH.
- Inhibition of VSV binding and infectivity by phosphatidylserine: is phosphatidylserine a VSV-binding site?
- An agar overlay plaque assay method for autographa californica nuclear-polyhedrosis virus
- Adsorptive endocytosis of Semliki Forest virus.
- The shape of vesicular stomatitis virus.
- Immunocytochemical localization of vesicular stomatitis virus proteins N and NS with monoclonal antibodies
- Evidence for biphasic uncoating during HIV-1 infection from a novel imaging assay
- Polykaryocyte formation induced by VSV in mouse L cells.
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