Iron accumulation in alcoholic liver diseases.

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Summary

The recent finding regarding the increase of pro-hepcidin serum in alcoholic patients with high serum ferritin support the assumption that a parenchymal iron deposition in mild ALD is an important factor to trigger hepatocytes injury by ethanol, and the possible cause of parencynalIron deposition may be an increase of cellular iron uptake via serum transferrin in hepatocytes after ethanol exposure.

Type
review
Published
2005-11-01
Cited by
101
References
38

Keywords

Transferrin, Transferrin receptor, Chemistry, Alcoholic liver disease, Oxidative stress

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