Compromised Humoral and Delayed-Type Hypersensitivity Responses in IL-23-Deficient Mice1
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Summary
IL-23 plays a critical role in T cell-dependent immune responses, and the data provide further support for the existence of an IL-23/IL-17 axis of communication between the adaptive and innate parts of the immune system.
- Type
- article
- Published
- 2004-03-01
- Cited by
- 224
- References
- 40
- Access
- Open access
- OpenAlex
- https://openalex.org/W1905324622
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:23419457
Keywords
Immune system, Immunology, Cytokine, Biology, Acquired immune system
References
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- IL-27, a heterodimeric cytokine composed of EBI3 and p28 protein, induces proliferation of naive CD4+ T cells.
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- Biology of recently discovered cytokines: Interleukin-17 – a unique inflammatory cytokine with roles in bone biology and arthritis
- Poly(inosinic-cytidylic) Acid–Triggered Exacerbation of Experimental Asthma Depends on IL-17A Produced by NK Cells
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- IL-23 Enhances the Inflammatory Cell Response in Cryptococcus neoformans Infection and Induces a Cytokine Pattern Distinct from IL-121
- Induction of immunity and inflammation by interleukin-12 family members.
- The Il-IL/23-17 Immune Pathway in Arthritis
- Interleukin‐23: as a drug target for autoimmune inflammatory diseases
- Tc17, a Unique Subset of CD8 T Cells That Can Protect against Lethal Influenza Challenge
- Nociceptive Sensory Fibers Drive Interleukin-23 Production from CD301b+ Dermal Dendritic Cells and Drive Protective Cutaneous Immunity