Possible coupling of prostaglandin E2 receptor with pertussis toxin-sensitive guanine nucleotide-binding protein in osteoblast-like cells.
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Summary
The results indicate the possible coupling of PGE2 signalling with pertussis toxin-sensitive GTP-binding protein, which is probably G12, in osteoblast-like cells.
- Type
- article
- Published
- 1991-02-01
- Cited by
- 24
- References
- 0
- OpenAlex
- https://openalex.org/W1834325518
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:20542244
Keywords
Pertussis toxin, G protein, GTP', ADP-ribosylation, Chemistry
References
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Cited by
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- Proliferative effect of PGD[2] on osteoblast-like cells : independent activation of pertussis toxin-sensitive GTP-binding protein from PGE[2] or PGF[2]α
- Effect of prostaglandin E2 on phospholipase D activity in osteoblast‐like MC3T3‐E1 cells
- Effects of endothelin‐1 on signal transduction in UMR‐106 osteoblastic cells
- Intracellular signaling mechanism of bradykinin in osteoblast-like cells: comparison with prostaglandin E2.
- Protein kinases A and C positively regulate G protein‐dependent activation of phosphatidylinositol‐specific phospholipase C by tumor necrosis factor‐alpha in MC3T3‐E1 osteoblasts
- Differential regulation of receptor‐stimulated cyclic adenosine monophosphate production by polyvalent cations in MC3T3‐E1 osteoblasts
- Effects of vitamin D3 on signaling by prostaglandin E2 in osteoblast‐like cells
- Effects of glucocorticoid on signalling by prostaglandin E2 in osteoblast-like cells.
- Effect of endothelin-1 on phospholipase D activity in osteoblast-like cells.
- Effect of Retinoic Acid on Prostaglandin F2α-Induced Prostaglandin E2 Synthesis in Osteoblast-Like Cells
- Cross-talk regulation between cyclic AMP production and phosphoinositide hydrolysis induced by prostaglandin E2 in osteoblast-like cells.
- Autoregulation of prostaglandin E2-induced Ca2+ influx in osteoblast-like cells: inhibition by self-induced activation of protein kinase C.
- Effects of retinoic acid on signalling by prostaglandin E2 in osteoblast-like cells.
- Inhibitory effect of 17 beta -estradiol on prostaglandin E2-induced phosphoinositide hydrolysis in osteoblast-like cells.
- Mechanism of prostaglandin E2-induced arachidonic acid release in osteoblast-like cells: independence from phosphoinositide hydrolysis.
- Mechanism of prostaglandin E2-stimulated heat shock protein 27 induction in osteoblast-like MC3T3-E1 cells.
- Endothelin-1 actions on resorption, collagen and noncollagen protein synthesis, and phosphatidylinositol turnover in bone organ cultures.
- Interleukin-6 synthesis induced by prostaglandin E2: cross-talk regulation by protein kinase C.
- Thrombin induces proliferation of osteoblast‐like cells through phosphatidylcholine hydrolysis
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