Expression of chemokine receptor CX3CR1 in infants with respiratory syncytial virus bronchiolitis
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Summary
Examination of the relationship between RSV infection and expression of perforin and IFN‐γ in CX3CR1‐expressing peripheral blood CD8+ T cells indicates existence of an event persisting after acute RSV infections that is able to modulate effector functions of cytotoxic T cells, and also link disease severity with CX 3CR1 expression.
- Type
- article
- Published
- 2008-03-01
- Cited by
- 13
- References
- 44
- OpenAlex
- https://openalex.org/W1490006635
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:18908584
Keywords
Perforin, Bronchiolitis, Immunology, Medicine, Chemokine
References
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- NF-κB regulation in the immune system
- Chemokine receptors and their role in inflammation and infectious diseases.
- CX3C chemokine mimicry by respiratory syncytial virus G glycoprotein
- Progressive Differentiation and Commitment of CD8+ T Cells to a Poorly Cytolytic CD8low Phenotype in the Presence of IL-41
- Fractalkine (CX3CL1) as an amplification circuit of polarized Th1 responses.
- Increased Toll‐like receptor 4 expression in infants with respiratory syncytial virus bronchiolitis
- A new class of membrane-bound chemokine with a CX3C motif
- Predominant type‐2 response in infants with respiratory syncytial virus (RSV) infection demonstrated by cytokine flow cytometry
- Respiratory syncytial virus infections in infants: quantitation and duration of shedding.
- Alternative Mechanisms of Respiratory Syncytial Virus Clearance in Perforin Knockout Mice Lead to Enhanced Disease
- Fractalkine (CX3CL1) stimulated by nuclear factor kappaB (NF-kappaB)-dependent inflammatory signals induces aortic smooth muscle cell proliferation through an autocrine pathway.
- Persistent infection of B lymphocytes by bovine respiratory syncytial virus.
- Identification and Molecular Characterization of Fractalkine Receptor CX3CR1, which Mediates Both Leukocyte Migration and Adhesion
- Persistence of respiratory syncytial virus genome and protein after acute bronchiolitis in guinea pigs.
- Expression of CX3CR1 chemokine receptors on neurons and their role in neuronal survival.
- Phosphorylation and down-regulation of CD4 and CD8 in human CTLs and mouse L cells
- Hospitalization for RSV bronchiolitis before 12 months of age and subsequent asthma, atopy and wheeze: A longitudinal birth cohort study
- Latency and persistence of respiratory syncytial virus despite T cell immunity.
- The G Glycoprotein of Respiratory Syncytial Virus Depresses Respiratory Rates through the CX3C Motif and Substance P
Cited by
- Immunological, Viral, Environmental, and Individual Factors Modulating Lung Immune Response to Respiratory Syncytial Virus
- CX3CR1 Is Expressed in Differentiated Human Ciliated Airway Cells and Co-Localizes with Respiratory Syncytial Virus on Cilia in a G Protein-Dependent Manner
- The role of cytokines and chemokines in severe respiratory syncytial virus infection and subsequent asthma.
- Innate immune response and bronchiolitis and preschool recurrent wheeze
- Decrease in circulating DNA, IL-10 and BAFF levels in newly-diagnosed SLE patients after corticosteroid and chloroquine treatment.
- Pattern recognition receptors for respiratory syncytial virus infection and design of vaccines.
- Hepatitis during respiratory syncytial virus infection – a case report
- Potent High-Affinity Antibodies for Treatment and Prophylaxis of Respiratory Syncytial Virus Derived from B Cells of Infected Patients
- Immune biomarkers predicting bronchiolitis disease severity: A systematic review.
- Biomarkers for Disease Severity in Children Infected With Respiratory Syncytial Virus: A Systematic Literature Review.
- Alterations in CX3CL1 Levels and Its Role in Viral Pathogenesis
- Respiratory syncytial virus glycoprotein G impedes CX3CR1-activation by CX3CL1 and monocyte function
- Infected Patients Syncytial Virus Derived from B Cells of Treatment and Prophylaxis of Respiratory Potent High-Affinity Antibodies for
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