Circumvention of drug resistance with calcium channel blockers and monoclonal antibodies.
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Summary
One of the major obstacles in current cancer chemotherapy is the development of drug resistance during treatment, and it has been found that tumor cells can acquire resistance to anticancer drugs.
- Type
- review
- Published
- 1989-01-01
- Cited by
- 14
- References
- 129
- OpenAlex
- https://openalex.org/W1026738519
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:1498158
Keywords
Drug resistance, Drug, Medicine, Pharmacology, Monoclonal antibody
References
- Dimethylsulfoxide, retinoic acid and 12-O-tetradecanoylphorbol-13-acetate induce a selective decrease in the phosphorylation of P150, a surface membrane phosphoprotein of HL60 cells resistant to adriamycin.
- Reversal of chloroquine resistance in Plasmodium falciparum by verapamil.
- Calcium and phosphatidylinositol turnover as signalling for transmembrane control of protein phosphorylation.
- Three multifunctional protein kinase systems in transmembrane control.
- Molecular Actions and Targets for Cancer Chemotherapeutic Agents
- Characteristics of resistance to adriamycin in human myelogenous leukemia K562 resistant to adriamycin and in isolated clones.
- ATP‐binding properties of P glycoprotein from multidrug‐resistant KB cells
- Anthracycline resistance in P388 murine leukemia and its circumvention by calcium antagonists.
- Effects of quinidine and related compounds on cytotoxicity and cellular accumulation of vincristine and adriamycin in drug-resistant tumor cells.
- Potentiation of etoposide-induced DNA damage by calcium antagonists in L1210 cells in vitro.
- Certain calcium channel blockers bind specifically to multidrug-resistant human KB carcinoma membrane vesicles and inhibit drug binding to P-glycoprotein.
- Mechanism of cross-resistance between vincristine and daunorubicin in Ehrlich ascites tumor cells.
- Enhancement by cyclosporin A of daunorubicin efficacy in Ehrlich ascites carcinoma and murine hepatoma 129.
- Pleiotropic drug resistance and gene amplification in a SEWA mouse tumor cell line. Complex relations revealed by drug uptake data, and lipid and protein analysis.
- Inhibitory action of chlorpromazine, dibucaine, and other phospholipid-interacting drugs on calcium-activated, phospholipid-dependent protein kinase.
- Purification of the 170- to 180-kilodalton membrane glycoprotein associated with multidrug resistance. 170- to 180-kilodalton membrane glycoprotein is an ATPase.
- Isolation and characterization of an anthracycline-resistant human leukemic cell line.
- ATP/Mg2+-dependent binding of vincristine to the plasma membrane of multidrug-resistant K562 cells.
- Overcoming of vincristine resistance in P388 leukemia in vivo and in vitro through enhanced cytotoxicity of vincristine and vinblastine by verapamil.
- Increased vinblastine binding to membrane vesicles from multidrug-resistant KB cells.
Cited by
- Relationship between multidrug resistant gene expression and multidrug resistant-reversing effect of MS-209 in various tumor cells
- In vitro models of multiple drug resistance.
- Multifunctional modulators of drug resistance.
- Function and regulation of the human multidrug resistance gene.
- Pharmacologic circumvention of multidrug resistance
- Cytotoxicity to macrophages of tetrandrine, an antisilicosis alkaloid, accompanied by an overproduction of prostaglandins.
- Induction of multidrug resistance gene expression during cholestasis in rats and nonhuman primates
- Reversal of multidrug resistance by a novel quinoline derivative, MS-209
- Reversal of multidrug resistance by an immunosuppressive agent FK-506
- P-glycoproteins and multidrug resistance.
- Multidrug resistance and anticonvulsants: new studies with some enaminones.
- Multidrug resistance in the laboratory and clinic.
- Vaccin therapeutique cible contre la p-glycoproteine 170 pour inhiber la resistance multidrogues dans le traitement des cancers
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