Effect of Cilazapril on Ventricular Remodeling Assessed by Doppler-Echocardiographic Assessment and Cardiac Gene Expression
Explore this paper's citation graph
Summary
Cilazapril improved the adverse remodeling process by attenuating the progression of systolic and diastolic dysfunction, and prevented abnormal cardiac gene expression following MI.
- Type
- article
- Published
- 1998-05-09
- Cited by
- 20
- References
- 53
- OpenAlex
- https://openalex.org/W230412782
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:5923232
Keywords
Cilazapril, Medicine, Internal medicine, Diastole, Cardiology
References
- Alterations in cardiac gene expression during the transition from stable hypertrophy to heart failure. Marked upregulation of genes encoding extracellular matrix components.
- Experimental myocardial infarction in the rat: qualitative and quantitative changes during pathologic evolution.
- Does converting enzyme inhibition change the neuronal and extraneuronal uptake of catecholamines?
- Coordinate regulation of the levels of type III and type I collagen mRNA in most but not all mouse fibroblasts.
- Developmental and functional adaptation of contractile proteins in cardiac and skeletal muscles.
- DNA synthesis in the non-infarcted cardiac interstitium after left coronary artery ligation in the rat: effects of captopril.
- Left ventricular remodeling after myocardial infarction: a corollary to infarct expansion.
- Importance of Venodilatation in Prevention of Left Ventricular Dilatation After Chronic Large Myocardial Infarction in Rats: A Comparison of Captopril and Hydralazine
- Amino-acid sequence of a Ca2++Mg2+-dependent ATPase from rabbit muscle sarcoplasmic reticulum, deduced from its complementary DNA sequence
- Regulation of angiotensin II receptors on ventricular myocytes after myocardial infarction in rats.
- Doppler-Derived Mitral Deceleration Time of Early Filling as a Strong Predictor of Pulmonary Capillary Wedge Pressure in Postinfarction Patients with Left Ventricular Systolic Dysfunction
- Pathological Hypertrophy and Cardiac Interstitium: Fibrosis and Renin‐Angiotensin‐Aldosterone System
- Inhibition by angiotensin II type 1 receptor antagonist of cardiac phenotypic modulation after myocardial infarction.
- Contribution of cardiac renin-angiotensin system to ventricular remodelling in myocardial-infarcted rats.
- Ventricular Remodeling After Myocardial Infarction: Experimental Observations and Clinical Implications
- Structural remodelling of the heart by fibrous tissue: role of circulating hormones and locally produced peptides.
- Role of angiotensin II in renal injury of deoxycorticosterone acetate-salt hypertensive rats.
- Prognostic value of Doppler transmitral flow patterns in patients with congestive heart failure.
- Effects of enalapril on mortality in severe congestive heart failure: results of the Cooperative North Scandinavian Enalapril Survival Study (CONSENSUS).
- Angiotensin converting enzyme and myofibroblasts during tissue repair in the rat heart.
Cited by
- Effect of Diltiazem on Cardiac Remodeling in Rats Assessed by Doppler Echocardiography and mRNA Expression
- Attenuation of changes in sarcoplasmic reticular gene expression in cardiac hypertrophy by propranolol and verapamil
- Advancing knowledge of right ventricular pathophysiology in chronic pressure overload: Insights from experimental studies.
- Coordinate Regulation of Metabolic Enzyme Encoding Genes During Cardiac Development and Following Carvedilol Therapy in Spontaneously Hypertensive Rats
- Partial prevention of changes in SR gene expression in congestive heart failure due to myocardial infarction by enalapril or losartan
- Transmitral inflow pattern assessed by Doppler echocardiography in angiotensin II type 1A receptor knockout mice with myocardial infarction.
- Sarcoplasmic reticulum Ca2+ transport and gene expression in congestive heart failure are modified by imidapril treatment.
- Pathophysiological Basis of Right Ventricular Remodeling
- Mechanisms of combined treatment with celiprolol and candesartan for ventricular remodeling in experimental heart failure.
- Nipradilol can prevent left ventricular systolic and diastolic dysfunction after myocardial infarction in rats.
- Comparative effects of cilazapril, carvedilol and their combination in preventing from left ventricular remodelling after acute myocardial infarction in rats.
- Hydrogen gas attenuates embryonic gene expression and prevents left ventricular remodeling induced by intermittent hypoxia in cardiomyopathic hamsters.
- The effect of sildenafil on right ventricular remodeling in a rat model of monocrotaline-induced right ventricular failure
- Right ventricular phenotype, function, and failure: a journey from evolution to clinics
- Role of angiotensin II in the development of subcellular remodeling in heart failure
- Mechanisms for the development of heart failure and improvement of cardiac function by angiotensin-converting enzyme inhibitors
- Molecular Mechanisms of Subcellular Remodeling in Congestive Heart Failure
- Hormonal Mechanisms of Cardiac Remodeling in Heart Failure
- Remodelage du Ventricule Droit dans l’Hypertension Pulmonaire Chronique Expérimentale
Related papers
- Type 2 diabetes and heart failure: Characteristics and prognosis in preserved, mid-range and reduced ventricular function
- Left Ventricular Function in Hospitalized Geriatric Patients
- Heart failure with preserved ejection fraction; the other half of the heart failure, how it stands in 2013
- Current Insights and Future Directions in the Treatment of Heart Failure with Preserved Ejection Fraction
- Abstract 13703: Outpatient Cardiac Rehabilitation Reduces Heart Failure Re-Hospitalization in Patient With Heart Failure With Reduced Ejection Fraction When Compared to Patients With Heart Failure With Preserved Ejection Fraction
- Age differences in contemporary treatment of patients with chronic heart failure and reduced ejection fraction
- Diagnosis and management of diastolic dysfunction and heart failure.
- Improvement of left ventricular diastolic dysfunction in hypertensive patients 1 month after ACE inhibition therapy: Evaluation by ultrasonic automated boundary detection
- Assessment of coronary flow reserve with transthoracic Doppler echocardiography: comparison with intracoronary Doppler method.