Melatonin Rescues Influenza A Virus–Induced Cellular Energy Exhaustion via OMA1‐OPA1‐S in Acute Exacerbation of COPD
Explore this paper's citation graph
Summary
Melatonin rescued IAV–induced cellular energy exhaustion via OMA1‐OPA1‐S to improve the clinical prognosis in COPD and may serve as a potential therapeutic agent for patients in which AECOPD is induced by IAV.
- Type
- article
- Published
- 2024-07-22
- Cited by
- 8
- References
- 76
- OpenAlex
- https://openalex.org/W39039850
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:271332637
Keywords
Computer science
References
- Salivary Melatonin as a Circadian Phase Marker: Validation and Comparison to Plasma Melatonin
- Resting energy expenditure in patients with chronic obstructive pulmonary disease.
- Respiratory infectious phenotypes in acute exacerbation of COPD: an aid to length of stay and COPD Assessment Test
- Upper-Respiratory Viral Infection, Biomarkers, and COPD Exacerbations
- Respiratory viruses, symptoms, and inflammatory markers in acute exacerbations and stable chronic obstructive pulmonary disease.
- Equations for the prediction of resting energy expenditure in chronic obstructive lung disease.
- Mitochondria in the regulation of innate and adaptive immunity
- The i-AAA protease YME1L and OMA1 cleave OPA1 to balance mitochondrial fusion and fission
- Antivirals for influenza: where now for clinical practice and pandemic preparedness?
- Viral Activation of Cellular Metabolism
- Epidemiological relationships between the common cold and exacerbation frequency in COPD
- Viral Etiology of Acute Exacerbations of COPD in Hong Kong
- Mitochondrial Cristae Shape Determines Respiratory Chain Supercomplexes Assembly and Respiratory Efficiency
- Procaterol inhibits rhinovirus infection in primary cultures of human tracheal epithelial cells.
- Association between mitochondrial dysfunction and severity and outcome of septic shock.
- Stress‐induced OMA1 activation and autocatalytic turnover regulate OPA1‐dependent mitochondrial dynamics
- Metabolic effects of influenza virus infection in cultured animal cells: Intra- and extracellular metabolite profiling
- Mitochondrial Dynamics and Metabolic Regulation.
- Metabolic regulation of mitochondrial dynamics
- Energy metabolic disorder is a major risk factor in severe influenza virus infection: Proposals for new therapeutic options based on animal model experiments.
Cited by
- Melatonin’s Impact on Wound Healing
- Microplastics exacerbate ferroptosis via mitochondrial reactive oxygen species-mediated autophagy in chronic obstructive pulmonary disease
- Melatonin protects against fluoride-induced developmental neurotoxicity by alleviating abnormal mitophagy and apoptosis via the PINK1/Parkin pathway.
- Melatonin inhibits ferroptosis by activating CREB-GPX4/ferritin axis in cigarette smoke-induced chronic obstructive pulmonary disease.
- Beyond a metabolite: lactate and lactylation in lung diseases
- Beyond pathogens: a narrative review of the immunological nexus of damage-associated molecular patterns and inflammasome activation in sterile AECOPD
Related papers
No related papers recorded.