Type I interferon does not promote susceptibility to foodborne Listeria monocytogenes
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Summary
The results highlight the importance of using the natural route of infection for studies of host–pathogen interactions and suggest that the detrimental effects of IFN-α/β signaling on the innate immune response to L. monocytogenes may be an artifact of the i.v. infection model.
- Type
- article
- Published
- 2016-02-19
- Cited by
- 32
- References
- 55
- OpenAlex
- https://openalex.org/W26895837
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:25645645
Keywords
Political science
References
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Cited by
- Type I Interferons in Bacterial Infections: A Balancing Act
- Apoptotic cells enhance pathogenesis of Listeria monocytogenes.
- Type I and III Interferon in the Gut: Tight Balance between Host Protection and Immunopathology
- Getting “Inside” Type I IFNs: Type I IFNs in Intracellular Bacterial Infections
- Down regulation of macrophage IFNGR1 exacerbates systemic L. monocytogenes infection
- Cyclic di-AMP in host-pathogen interactions.
- Type I interferon signaling restrains IL-10R+ colonic macrophages and dendritic cells and leads to more severe Salmonella colitis
- Neutrophils from Both Susceptible and Resistant Mice Efficiently Kill Opsonized Listeria monocytogenes
- A Comparison of Oral and Intravenous Mouse Models of Listeriosis
- THE ROLE OF PRO-INFLAMMATORY MEDIATORS IFNβ AND PROSTAGLANDIN E2 IN SUPPRESSION OF INNATE IMMUNITY TO LISTERIA MONOCYTOGENES
- Sources of Type I Interferons in Infectious Immunity: Plasmacytoid Dendritic Cells Not Always in the Driver's Seat
- Emerging Roles of Autophagy and Inflammasome in Ehrlichiosis
- Innate and Adaptive Immune Responses during Listeria monocytogenes Infection
- Ligand-induced IFNGR1 down-regulation calibrates myeloid cell IFNγ responsiveness
- Select autophagy genes maintain quiescence of tissue-resident macrophages and increase susceptibility to Listeria monocytogenes
- Pathogenic Biohacking: Induction, Modulation and Subversion of Host Transcriptional Responses by Listeria monocytogenes
- Enrichment of neutrophils and monocytes from the liver following either oral or intravenous Listeria monocytogenes infection
- Secretion of c-di-AMP by Listeria monocytogenes Leads to a STING-Dependent Antibacterial Response during Enterocolitis
- Context Is Key: Delineating the Unique Functions of IFNα and IFNβ in Disease
- MicroRNA-30e-5p Regulates SOCS1 and SOCS3 During Bacterial Infection
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