H2S restores the cardioprotection from ischemic post‐conditioning in isolated aged rat hearts
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Summary
It is suggested that exogenous H2S recovers PC‐induced cardioprotection via MAPK pathway in the aged hearts through the ischemia/reperfusion mechanism.
- Type
- article
- Published
- 2015-10-01
- Cited by
- 12
- References
- 12
- OpenAlex
- https://openalex.org/W26113037
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:27834193
Keywords
Environmental science
References
- Preconditioning and postconditioning: united at reperfusion.
- Exogenous hydrogen sulfide postconditioning protects isolated rat hearts against ischemia-reperfusion injury.
- Aging mouse hearts are refractory to infarct size reduction with post-conditioning.
- H2S as a Physiologic Vasorelaxant: Hypertension in Mice with Deletion of Cystathionine γ-Lyase
- Interaction of Hydrogen Sulfide and Estrogen on the Proliferation of Vascular Smooth Muscle Cells
- Mediation of exogenous hydrogen sulfide in recovery of ischemic post-conditioning-induced cardioprotection via down-regulating oxidative stress and up-regulating PI3K/Akt/GSK-3β pathway in isolated aging rat hearts
- Cystathionine γ-lyase protects against renal ischemia/reperfusion by modulating oxidative stress.
- Postconditioning: reduction of reperfusion-induced injury.
- Endogenous hydrogen sulphide mediates the cardioprotection induced by ischemic postconditioning.
- Hydrogen sulfide‐induced apoptosis of human aorta smooth muscle cells via the activation of mitogen‐activated protein kinases and caspase‐3
- Loss of cardioprotection with ageing.
- Involvement of dopamine D2 receptors activation in ischemic post-conditioning-induced cardioprotection through promoting PKC-ε particulate translocation in isolated rat hearts
Cited by
- Exogenous H2S contributes to recovery of ischemic post-conditioning-induced cardioprotection by decrease of ROS level via down-regulation of NF-κB and JAK2-STAT3 pathways in the aging cardiomyocytes
- Involvement of exogenous H2S in recovery of cardioprotection from ischemic post-conditioning via increase of autophagy in the aged hearts.
- Protective Effects of Hydrogen Sulfide in the Ageing Kidney
- Impaired RISK-GSK3β Pathway is Responsible for Comorbidities by Suppressing the Conditioning Cardioprotection
- Exogenous H2S restores ischemic post-conditioning-induced cardioprotection through inhibiting endoplasmic reticulum stress in the aged cardiomyocytes
- Remifentanil preconditioning confers cardioprotection via c‐Jun NH2‐terminal kinases and extracellular signal regulated kinases pathways in ex‐vivo failing rat heart
- c-Jun N-Terminal Kinases (JNKs) in Myocardial and Cerebral Ischemia/Reperfusion Injury
- H2S restores the cardioprotective effects of ischemic post-conditioning by upregulating HB-EGF/EGFR signaling
- Involvement of HSP90 in ischemic postconditioning-induced cardioprotection by inhibition of the complement system, JNK and inflammation
- Fighting Oxidative Stress with Sulfur: Hydrogen Sulfide in the Renal and Cardiovascular Systems
- Protective Mechanism of Humanin Against Oxidative Stress in Aging-Related Cardiovascular Diseases
- Diabetes Worsens Outcomes After Asphyxial Cardiac Arrest in Rats
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