Drosophila Erect wing (Ewg) controls mitochondrial fusion during muscle growth and maintenance by regulation of the Opa1-like gene
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Summary
It is shown in Drosophila indirect flight muscles that the nuclear-encoded mitochondrial inner membrane fusion gene, Opa1-like, is regulated in a spatiotemporal fashion by the transcription factor/co-activator Erect wing (Ewg).
- Type
- article
- Published
- 2014-01-01
- Cited by
- 36
- References
- 76
- OpenAlex
- https://openalex.org/W24198395
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:12674540
Keywords
Philosophy, Derecho, Humanities
References
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- Down‐regulation of mitochondrial mRNAs in the mdx mouse model for Duchenne muscular dystrophy
- Expression Profiling in the Muscular Dystrophies
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- Loss of OPA1 Perturbates the Mitochondrial Inner Membrane Structure and Integrity, Leading to Cytochrome c Release and Apoptosis*
- Characterisation of missense mutations in the Act88F gene of Drosophila melanogaster
- The effect of aging on human skeletal muscle mitochondrial and intramyocellular lipid ultrastructure.
- Conserved Regions of the Drosophila Erect Wing Protein Contribute Both Positively and Negatively to Transcriptional Activity*
- The Drosophila PGC‐1 homologue Spargel coordinates mitochondrial activity to insulin signalling
- Differences in mitochondrial movement and morphology in young and mature primary cortical neurons in culture.
- The PINK1-Parkin pathway is involved in the regulation of mitochondrial remodeling process.
Cited by
- E2F function in muscle growth is necessary and sufficient for viability in Drosophila
- Spatio-temporal coordination of cell cycle exit, fusion and differentiation of adult muscle precursors by Drosophila Erect wing (Ewg).
- Drosophila expressing human SOD1 successfully recapitulates mitochondrial phenotypic features of familial amyotrophic lateral sclerosis.
- Differential Protein Distribution between the Nucleus and Mitochondria: Implications in Aging
- Identification and functional characterization of muscle satellite cells in Drosophila
- Metformin restores the mitochondrial network and reverses mitochondrial dysfunction in Down syndrome cells
- Overexpression of miRNA-9 Generates Muscle Hypercontraction Through Translational Repression of Troponin-T in Drosophila melanogaster Indirect Flight Muscles
- Honey protects against wings posture error and molecular changes related to mitochondrial pathways induced by hypoxia/reoxygenation in adult Drosophila melanogaster.
- Suppression of myopathic lamin mutations by muscle‐specific activation of AMPK and modulation of downstream signaling
- Essential roles of mitochondrial biogenesis regulator Nrf1 in retinal development and homeostasis
- Protein composition of the muscle mitochondrial reticulum during postnatal development
- Pioglitazone Improves Mitochondrial Organization and Bioenergetics in Down Syndrome Cells
- Fundc1 is necessary for proper body axis formation during embryogenesis in zebrafish
- Targeting Mitochondrial Network Architecture in Down Syndrome and Aging
- Indirect flight muscles in Drosophila melanogaster as a tractable model to study muscle development and disease.
- Myofibril and mitochondria morphogenesis are coordinated by a mechanical feedback mechanism in muscle
- The Role of Muscle in Insect Energy Homeostasis
- The Functional Impact of Mitochondrial Structure Across Subcellular Scales
- Marf-mediated mitochondrial fusion is imperative for the development and functioning of indirect flight muscles (IFMs) in Drosophila.
- Mitochondrial Dynamics in the Drosophila Ovary Regulates Germ Stem Cell Number, Cell Fate, and Female Fertility
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