Lewy bodies in progressive supranuclear palsy
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Summary
This study suggests that patients who have PSP with LBs constitute a subset of patients with PSP in whom Lewy body disease is also present, and double-labeling immunohistochemical studies showed co-localization of α-synuclein and tau in some neurons.
- Type
- article
- Published
- 2002-05-04
- Cited by
- 62
- References
- 27
- OpenAlex
- https://openalex.org/W20642932
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:24919878
Keywords
Progressive supranuclear palsy, Locus coeruleus, Lewy body, Pathology, Reticular formation
References
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- Progressive supranuclear palsy
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Cited by
- Cortical Alzheimer type pathology does not influence tau pathology in progressive supranuclear palsy.
- Autosomal recessive, early-onset Parkinson’s disease
- The Synaptic Function of α-Synuclein
- Coexistence of PSP and MSA: a case report and review of the literature
- A critical review of the development and importance of proteinaceous aggregates in animal models of Parkinson's disease: new insights into Lewy body formation.
- ELEVATED TAUOPATHY AND ALPHA SYNUCLEIN PATHOLOGY IN POSTMORTEM PARKINSON’S DISEASE BRAINS WITH AND WITHOUT DEMENTIA
- The Relationship between Parkin and Protein Aggregation in Neurodegenerative Diseases
- Tuft-shaped astrocytes in Lewy body disease
- Lewy Bodies in Progressive Supranuclear Palsy Represent an Independent Disease Process
- The mitochondrial complex I inhibitor rotenone triggers a cerebral tauopathy
- Identification of G-Protein Coupled Receptor Kinase 2 in Paired Helical Filaments and Neurofibrillary Tangles
- Biomarker identification in neurologic diseases: improving diagnostics and therapeutics
- Is there a role for naturally occurring cyanobacterial toxins in neurodegeneration? The beta-N-methylamino-L-alanine (BMAA) paradigm.
- Tauopathies and synucleinopathies: Do cerebrospinal fluid β-amyloid peptides reflect disease-specific pathogenesis?
- Co-existence of Parkinson’s disease and progressive supranuclear palsy: case report and a review of the literature
- Clinical Features of Patients with Concomitant Parkinson's Disease and Progressive Supranuclear Palsy Pathology
- The neurotoxin, MPP+, induces hyperphosphorylation ofTau, in the presence of α-Synuclein, in SH-SY5Y neuroblastoma cells
- Midbrain catecholaminergic neurons co-express α-synuclein and tau in progressive supranuclear palsy
- Different Tau Pathology Pattern in Two Clinical Phenotypes of Progressive Supranuclear Palsy
- Interface between tauopathies and synucleinopathies: A tale of two proteins
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