Regulation und Funktion lymphozytärer Ionenkanäle – CRAC und ORCC – in Proliferation und Apoptose
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- Type
- dissertation
- Published
- 2002-01-01
- Cited by
- 0
- References
- 174
- OpenAlex
- https://openalex.org/W18183881
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:81770817
Keywords
Biology, Molecular biology, Chemistry, Cell biology
References
- Caenorhabditis elegans gene ced-9 protects cells from programmed cell death
- Participation of target Fas protein in apoptosis pathway induced by CD4+ Th1 and CD8+ cytotoxic T cells.
- Store depletion and calcium influx.
- Bcl-2 blocks loss of mitochondrial membrane potential while ICE inhibitors act at a different step during inhibition of death induced by respiratory chain inhibitors.
- Bcl‐2 mutants with restricted subcellular location reveal spatially distinct pathways for apoptosis in different cell types.
- T cell receptor-mediated signaling is defective in T cells obtained from patients with primary intracranial tumors.
- Regulation by sphingomyelinase and sphingosine of Ca2+ signals elicited by CD3 monoclonal antibody, thapsigargin, or ionomycin in the Jurkat T cell line.
- PKC zeta is a molecular switch in signal transduction of TNF‐alpha, bifunctionally regulated by ceramide and arachidonic acid.
- Effect of gamma radiation on resting B lymphocytes. I. Oxygen-dependent damage to the plasma membrane results in increased permeability and cell enlargement.
- Tyrosine kinase activation provides an early and requisite signal for Fas-induced apoptosis.
- Submicromolar La3+ concentrations block the calcium release-activated channel, and impair CD69 and CD25 expression in CD3- or thapsigargin-activated Jurkat cells.
- Activation of Ca2+ current in Jurkat T cells following the depletion of Ca2+ stores by microsomal Ca(2+)-ATPase inhibitors.
- A novel protein domain required for apoptosis. Mutational analysis of human Fas antigen.
- Protein tyrosine phosphorylation is involved in osmoregulation of ionic conductances.
- Ceramide activates heterotrimeric protein phosphatase 2A.
- Cross-linking of the Fas/APO-1 antigen suppresses the CD3-mediated signal transduction events in human T lymphocytes.
- Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC
- Bcl-2 prolongs cell survival after Bax-induced release of cytochrome c
- Cytotoxicity‐dependent APO‐1 (Fas/CD95)‐associated proteins form a death‐inducing signaling complex (DISC) with the receptor.
- Both CFTR and outwardly rectifying chloride channels contribute to cAMP-stimulated whole cell chloride currents.
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