Cellular mechanisms associated with intermittent hypoxia.
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Summary
It is revealed that for a given duration and intensity, IH is more potent than continuous hypoxia in evoking transcriptional activation and ROS-mediated signalling mechanisms contribute to cellular and systemic responses to IH.
- Type
- article
- Published
- 2007-08-10
- Cited by
- 44
- References
- 54
- OpenAlex
- https://openalex.org/W17705795
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:44030880
Keywords
Political science, Humanities, Art
References
- Intracellular pathways linking hypoxia to activation of c-fos and AP-1.
- Sleep apnea, norepinephrine-release rate, and daytime hypertension.
- Proteomic analysis of CA1 and CA3 regions of rat hippocampus and differential susceptibility to intermittent hypoxia
- Role of oxidative stress in intermittent hypoxia‐induced immediate early gene activation in rat PC12 cells
- Chronic intermittent hypoxia induces hypoxia‐evoked catecholamine efflux in adult rat adrenal medulla via oxidative stress
- Systemic, cellular and molecular analysis of chemoreflex‐mediated sympathoexcitation by chronic intermittent hypoxia
- Chronic intermittent hypoxia decreases the expression of Na/H exchangers and HCO3-dependent transporters in mouse CNS.
- Serum levels of vascular endothelial growth factor are elevated in patients with obstructive sleep apnea and severe nighttime hypoxia.
- Regulation of hypoxia-inducible factor 1 by prolyl and asparaginyl hydroxylases.
- Asparagine Hydroxylation of the HIF Transactivation Domain: A Hypoxic Switch
- Effects of incomplete ionization of impurities in poly-Si gate and band gap narrowing on direct tunneling gate leakage current
- Increased oxidative stress is associated with chronic intermittent hypoxia-mediated brain cortical neuronal cell apoptosis in a mouse model of sleep apnea.
- Impaired physiological responses to chronic hypoxia in mice partially deficient for hypoxia-inducible factor 1alpha.
- Perspectives on oxygen sensing.
- Ca2+/Calmodulin Kinase-dependent Activation of Hypoxia Inducible Factor 1 Transcriptional Activity in Cells Subjected to Intermittent Hypoxia*
- Expression of c-fos in the rat brainstem after chronic intermittent hypoxia.
- Aconitase is a sensitive and critical target of oxygen poisoning in cultured mammalian cells and in rat lungs.
- Proteomic identification of a novel protein regulated in CA1 and CA3 hippocampal regions during intermittent hypoxia.
- Chronic intermittent hypoxia activates nuclear factor-kappaB in cardiovascular tissues in vivo.
- Intermittent hypoxic exposure during light phase induces changes in cAMP response element binding protein activity in the rat CA1 hippocampal region: water maze performance correlates.
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- Chronic Intermittent Hypoxia/Reoxygenation Facilitate Amyloid-β Generation in Mice
- Intermittent hypoxia during sleep induces reactive gliosis and limited neuronal death in rats: implications for sleep apnea
- Melatonin reduces hippocampal beta-amyloid generation in rats exposed to chronic intermittent hypoxia.
- Exogenous expression of HIF-1 alpha promotes cardiac differentiation of embryonic stem cells.
- Distinct Mechanisms Underlying Tolerance to Intermittent and Constant Hypoxia in Drosophila melanogaster
- ‘Combining Hypoxic Methods for Peak Performance’: a Biomedical Engineering Perspective
- Review: Hypoxic and oxidative stress resistance in Drosophila melanogaster
- Down-regulation of bile acid synthesis and a metabolic co-activator under hypoxic conditions - implications in obstructive sleep apnea.
- POST-TRANSLATIONAL MODIFICATION OF PROTEINS DURING INTERMITTENT HYPOXIA
- Modeling premature brain injury and recovery
- The Effect of Altitude-Induced Hypoxia on Heart Disease: Do Acute, Intermittent, and Chronic Exposures Provide Cardioprotection?
- Hypoxia and adipose tissue function and dysfunction in obesity.
- Hypoxia and the endocrine and signalling role of white adipose tissue
- Development of a pluripotent stem cell derived neuronal model to identify chemically induced pathway perturbations in relation to neurotoxicity: effects of CREB pathway inhibition.
- Hypoxic/ischemic conditions induce expression of the putative pro-death gene Clca1 via activation of extrasynaptic N-methyl-D-aspartate receptors.
- Apparent PKA activity responds to intermittent hypoxia in bone cells: a redox pathway?
- Molecular pathways of spontaneous and TNF-alpha-mediated neutrophil apoptosis under intermittent hypoxia.
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