Up-regulation of advanced glycated products receptors in the brain of diabetic rats is prevented by antioxidant treatment.
Explore this paper's citation graph
Summary
When dehydroepiandrosterone was administered to diabetic rats, the improved oxidative imbalance and the marked reduction of AGE receptors paralleled the reduced activation of nuclear factor kappaB and the reduction of S-100 levels, reinforcing the suggestion that oxidative stress plays a role in diabetes-related neuronal damage.
- Type
- article
- Published
- 2005-12-01
- Cited by
- 75
- References
- 62
- OpenAlex
- https://openalex.org/W16166220
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:22982248
Keywords
Geography
References
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding.
- THE BIOSYNTHESIS OF POLYSACCHARIDES. INCORPORATION OF D-(1-14C)GLUCOSE AND D-(6-14C)GLUCOSE INTO PLUM-LEAF POLYSACCHARIDES.
- Animal models and molecular markers for cerebral ischemia-reperfusion injury in brain.
- Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage
- Aldose reductase induction: a novel response to oxidative stress of smooth muscle cells
- The human and rat recombinant receptors for advanced glycation end products have a high degree of homology but different pharmacokinetic properties in rats.
- Diabetes mellitus in midlife and the risk of dementia three decades later
- Blockade of receptor for advanced glycation end product (RAGE) attenuates ischemia and reperfusion injury to the liver in mice
- Accelerated age-related browning of human collagen in diabetes mellitus.
- The role of oxidative stress in the onset and progression of diabetes and its complications: asummary of a Congress Series sponsored byUNESCO‐MCBN, the American Diabetes Association and the German Diabetes Society
- Diabetes mellitus and Cerebrovascular Disease
- Dehydroepiandrosterone modulates nuclear factor-kappaB activation in hippocampus of diabetic rats.
- Effect of treatment of diabetic rats with dehydroepiandrosterone on vascular and neural function.
- Inhibition of GAPDH activity by poly(ADP-ribose) polymerase activates three major pathways of hyperglycemic damage in endothelial cells.
- Protective effect of dehydroepiandrosterone against copper-induced lipid peroxidation in the rat.
- Aldose reductase from human tissues.
- Molecular Mechanisms of Tumor Necrosis Factor α Gene Expression in Monocytic Cells via Hyperglycemia-induced Oxidant Stress-dependent and -independent Pathways*
- Coregulation of Neurite Outgrowth and Cell Survival by Amphoterin and S100 Proteins through Receptor for Advanced Glycation End Products (RAGE) Activation*
- Using and understanding medical statistics
- Brain Metabolic Alterations in Patients with Type 1 Diabetes–Hyperglycemia-Induced Injury
Cited by
- Upregulation of galectin-3 in immortalized Schwann cells IFRS1 under diabetic conditions.
- Diabetes-Mediated Exacerbation of Neuronal Damage and Inflammation After Cerebral Ischemia in Rat: Protective Effects of Water-Soluble Extract from Culture Medium of Ganoderma lucidum Mycelia
- Evidence for Cortical Neuronal Loss in Male Type 2 Diabetic Goto-Kakizaki Rats
- Hepato‐/reno‐protective activity of Chinese prescription Kangen‐karyu through inhibition of AGE formation and fibrosis‐related protein expression in type 2 diabetes
- Growth Factor Receptor‐Bound Protein 10‐Mediated Negative Regulation of the Insulin‐Like Growth Factor‐1 Receptor‐Activated Signalling Pathway Results in Cognitive Disorder in Diabetic Rats
- Probenecid Treatment Enhances Retinal and Brain Delivery of N-4-Benzoylaminophenylsulfonylglycine, An Anionic Aldose Reductase Inhibitor
- Modulations of the calcineurin/NF-AT pathway in skeletal muscle atrophy.
- Beneficial effects of Chinese prescription Kangen-karyu on diabetes associated with hyperlipidemia, advanced glycation endproducts, and oxidative stress in streptozotocin-induced diabetic rats.
- Receptor for advanced glycation end products and neuronal deficit in the fatal brain edema of diabetic ketoacidosis.
- Skin fibroblast model to study an impaired glutathione synthesis: consequences of a genetic polymorphism on the proteome.
- Protective effects of grape seed proanthocyanidin extracts on cerebral cortex of streptozotocin-induced diabetic rats through modulating AGEs/RAGE/NF-kappaB pathway.
- Benfotiamine exhibits direct antioxidative capacity and prevents induction of DNA damage in vitro
- Lifestyle intervention reversed cognitive function in aged people with diabetes mellitus: two-year follow up.
- Cognitive dysfunction and diabetes mellitus.
- Cardiac impairment in rabbits fed a high-fat diet is counteracted by dehydroepiandrosterone supplementation.
- Oxidative stress-dependent impairment of cardiac-specific transcription factors in experimental diabetes.
- Immortalized Adult Rodent Schwann Cells as In Vitro Models to Study Diabetic Neuropathy
- Lutein and docosahexaenoic acid prevent cortex lipid peroxidation in streptozotocin-induced diabetic rat cerebral cortex.
- Prevention of severe toxic liver injury and oxidative stress in MCP-1-deficient mice.
- Paraoxonase 1 (PON1) attenuates diabetes development in mice through its antioxidative properties.
Related papers
No related papers recorded.