The non-selective voltage-activated cation channel in the human red blood cell membrane: reconciliation between two conflicting reports and further characterisation.
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Summary
Using the patch-clamp technique, the non-selective, voltage-activated cation channel in the human red blood cell (RBC) membrane was further characterised and demonstrated to be permeable to the divalent cations Ca2+ and Ba2+, and even Mg2+.
- Type
- dissertation
- Published
- 2000-12-01
- Cited by
- 109
- References
- 16
- Access
- Open access
- OpenAlex
- https://openalex.org/W11129235
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:21529132
Keywords
Coupon, Professional development, Public relations, Political science, Career development
References
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- Evidence for a voltage-gated, non-selective cation channel in the human red cell membrane.
- Stochastic nature and red cell population distribution of the sickling-induced Ca2+ permeability.
- Net Synthesis of ATP by Reversal of the Sodium Pump
- Slow voltage-dependent changes in channel open-state probability underlie hysteresis of NMDA responses in Mg(2+)-free solutions.
- Non-selective voltage-activated cation channel in the human red blood cell membrane.
- Regulation of Cell Volume by Active Cation Transport in High and Low Potassium Sheep Red Cells
- The Passive Permeability of the Red Blood Cell to Cations
- Increased choline transport in erythrocytes from mice infected with the malaria parasite Plasmodium vinckei vinckei.
- A novel pathway for Ca++ entry into Plasmodium falciparum-infected blood cells.
- Voltage-activated cation transport in human erythrocytes.
- Calcium transport and compartment analysis of free and exchangeable calcium in Plasmodium falciparum-infected red blood cells.
- The fifth discipline, the art and practice of the learning organization
- Nature of Prejudice
- The relationship of training motivation to participation in training and development
- Social loafing: A meta-analytic review and theoretical integration.
Cited by
- Pharmacology of the human red cell voltage-dependent cation channel; Part I. Activation by clotrimazole and analogues.
- Band 3 Missense Mutations and Stomatocytosis: Insight into the Molecular Mechanism Responsible for Monovalent Cation Leak
- Quantification of red blood cell adhesion using holographic optical tweezers and single cell force spectroscopy
- Identification of signalling cascades involved in red blood cell shrinkage and vesiculation
- Passive transport pathways for Ca(2+) and Co(2+) in human red blood cells. (57)Co(2+) as a tracer for Ca(2+) influx.
- Erythrocyte ion channels in regulation of apoptosis.
- The Regulatory Role of Temperature and Endothelin-B Receptor in Erythrocyte Programmed Cell Death
- Protein Kinase-Regulated Inwardly Rectifying Anion and Organic Osmolyte Channels in Malaria-Infected Erythrocytes
- Caractérisation des canaux anioniques dans la membrane du globule rouge humain infecté par Plasmodium falciparum
- Phosphatidylserine exposure in red blood cells : a suggestion for the active role of red blood cells in blood clot formation
- Conductance hysteresis in the voltage dependent anion-selective channel
- Dynamic memory of a single voltage-gated potassium ion channel: A stochastic nonequilibrium thermodynamic analysis.
- Effect of transmembrane potential on the diffusion of Na⁺/H⁺ exchanger of human red blood cell
- Patch-clamp analysis of the "new permeability pathways" in malaria-infected erythrocytes.
- Hysteresis in Channel Gating
- Der nicht-genomische Effekt des Mineralokortikoids Aldosteron auf die Transportsysteme der Erythrozytenmembran
- Vanadate-Induced Suicidal Erythrocyte Death
- Pharmacology of the human red cell voltage-dependent cation channel. Part II: inactivation and blocking.
- Resonant effects in a voltage-activated channel gating
- Carbon Monoxide–Sensitive Apoptotic Death of Erythrocytes
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