Modulation of Natural Killer Cell Cytotoxicity in Human Cytomegalovirus Infection: The Role of Endogenous Class I Major Histocompatibility Complex and a Viral Class I Homolog
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Summary
The role of UL18 in NK cell recognition and lysis is examined using fibroblasts infected with either wild-type or UL18 knockout CMV virus, and by using cell lines transfected with the UL18 gene.
- Type
- article
- Published
- 1998-05-18
- Cited by
- 216
- References
- 36
- OpenAlex
- https://openalex.org/W9584146
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:3774142
Keywords
Computer science
References
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Cited by
- Interactions of LIRs, a family of immunoreceptors expressed in myeloid and lymphoid cells, with viral and cellular MHC class I antigens.
- Surface expression of HLA-E, an inhibitor of natural killer cells, enhanced by human cytomegalovirus gpUL40.
- Exploitation of cellular signaling and regulatory pathways by human cytomegalovirus.
- MHC Class I-Independent Recognition of NK-Activating Receptor KIR2DS41
- NK cell receptors involved in the response to human cytomegalovirus infection.
- Function of CMV-encoded MHC class I homologues.
- Fatal attraction: cytomegalovirus-encoded chemokine homologs.
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- Viral Proteins Counteracting Host Defenses
- Caractérisation de l'activité fonctionnelle et métabolique des cellules NK en situation de stress nutritionnels : approche expérimentale in vitro et in vivo
- Viral and host gene expression during human cytomegalovirus infection.
- The immune response to cytomegalovirus and Epstein-Barr virus in systemic lupus erythematosus
- Dissecting the Structural Determinants of the Interaction between the Human Cytomegalovirus UL18 Protein and the CD85j Immune Receptor1
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