Low concentrations of nitric oxide increase oxygen affinity of sickle erythrocytes in vitro and in vivo.
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Summary
Low concentrations of NO gas may offer an attractive new therapeutic model for the treatment of SCD by augmenting the oxygen affinity of sickle erythrocytes in vitro and in vivo without significant Mhb production.
- Type
- article
- Published
- 1997-09-01
- Cited by
- 147
- References
- 33
- Access
- Open access
- OpenAlex
- https://openalex.org/W9276736
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:15929628
Keywords
Crying, Population, Psychology, Traumatic stress, Psychiatry
References
- The effect of carbon monoxide on red cell life span in sickle cell disease.
- Hemoglobin: Molecular, Genetic and Clinical Aspects
- Gelation of sickle cell haemoglobin. II. Methaemoglobin.
- Cooperativity in the dissociation of nitric oxide from hemoglobin.
- Covalent binding of glutathione to hemoglobin. II. Functional consequences and structural changes reflected in NMR spectra.
- Antisickling effects of 2,3-diphosphoglycerate depletion.
- Deoxygenated sickle hemoglobin. Modulation of its solubility by 2,3-diphosphoglycerate and other allosteric polyanions.
- Covalent binding of glutathione to hemoglobin. I. Inhibition of hemoglobin S polymerization.
- Sickle cell hemoglobin polymerization.
- Hemoglobin S gelation and sickle cell disease.
- Inhaled Nitric Oxide: A Selective Pulmonary Vasodilator Reversing Hypoxic Pulmonary Vasoconstriction
- Oxygen affinity as an index of hemoglobin S polymerization: a new micromethod.
- Hb Okazaki [β93(F8) Cys → Arg], a new hemoglobin variant with increased oxygen affinity and instability
- Nitric oxide can modify amino acid residues in proteins.
- Inhaled Nitric Oxide as a Cause of Selective Pulmonary Vasodilatation in Pulmonary Hypertension
- Selective Pulmonary Vasodilation by Inhaled Nitric Oxide Is Due to Hemoglobin Inactivation
- Intracellular Polymerization
- Effect of nitric oxide on the oxygen transport of human erythrocytes.
- Reaction of nitric oxide with heme proteins and model compounds of hemoglobin.
- Requirements for therapeutic inhibition of sickle haemoglobin gelation
Cited by
- Inhaled Nitric Oxide Delivery and Monitoring
- Sickle cell disease and aortic valve replacement: use of cardiopulmonary bypass, partial exchange transfusion, platelet sequestration, and continuous hemofiltration.
- The Effect of Nitric Oxide on Pain From Acute Vascular Occlusion Crisis in Sickle Cell Disease: A Systematic Review
- Non-invasive detection of endothelial dysfunction in sickle cell disease by Doppler ultrasonography.
- New advances in the treatment of sickle cell disease: focus on perioperative significance.
- Inhaled nitric oxide: Current clinical concepts.
- Effects of nitric oxide on red blood cell development and phenotype.
- Les effets bénéfiques de l'hydroxyurée dans l'anémie falciforme de l'enfant
- Inhaled nitric oxide for acute chest syndrome in people with sickle cell disease.
- Acute chest syndrome in sickle cell disease
- Extrapulmonary Effects of Inhaled Nitric Oxide
- The acute chest syndrome in sickle cell disease. Possible role of nitric oxide in its pathophysiology and treatment.
- In Vitro Impact of Treatment With Aqueous Extract of Cassia Fistula on Red Blood Cell Sickling in Individuals With Sickle Cell Trait
- Acute chest syndrome: sickle cell disease
- Detection of nitrosyl hemoglobin in venous blood in the treatment of sickle cell anemia with hydroxyurea.
- NO-body's perfect.
- Nitric oxide: Clinical applications
- Protein Glutathionylation in Cardiovascular Diseases
- Developing treatment for sickle cell disease
- Beneficial effects of nitric oxide breathing in adult patients with sickle cell crisis
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