Epithelial autotoxicity of nitric oxide: role in the respiratory cytopathology of pertussis.
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Summary
Nitric oxide mediates the toxicity of TCT for the respiratory epithelium, thus implicating a central role for nitric oxide in the pathogenesis of pertussis.
- Type
- article
- Published
- 1994-01-04
- Cited by
- 158
- References
- 0
- Access
- Open access
- OpenAlex
- https://openalex.org/W7506415
- Semantic Scholar
- https://api.semanticscholar.org/CorpusID:3074286
Keywords
Computer science, Massively parallel, Exploit, Digital electronics, Automatic test pattern generation
References
No references recorded for this paper.
Cited by
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- Genetics of virulence in Bordetella pertussis. I. Characterization of genes for superoxide dismutase and catalase. II. Identification of a new putative transcriptional regulator.
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- T cells and fibrosis.
- Nitric Oxide Synthase Inhibition
- Protein thiol modification and apoptotic cell death as cGMP-independent nitric oxide (NO) signaling pathways.
- Pertussis: a matter of immune modulation.
- The yin and yang of nitric oxide: Reflections on the physiology and pathophysiology of NO·
- Human lung mononuclear cells induce nitric oxide synthase in murine airway epithelial cells in vitro: role of TNFalpha and IL-1beta.
- Streptomyces spores from mouldy houses induce nitric oxide, TNFα and IL-6 secretion from RAW264.7 macrophage cell line without causing subsequent cell death.
- Activation of inducible nitric oxide synthase by Euonymus alatus in mouse peritoneal macrophages.
- Induction of nitric oxide synthase by Oldenlandia diffusa in mouse peritoneal macrophages.
- Signalling and cellular specificity of airway nitric oxide production in pertussis
- Meconium Enhances the Release of Nitric Oxide in Human Airway Epithelial Cells
- Nitric oxide synthases: roles, tolls, and controls.
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