A severe combined immunodeficiency mutation in the mouse

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Summary

Mice homozygous for this mutation have few if any lymphocytes; consequently they are hypogammaglobulinaemic and deficient for immune functions mediated by T and B lymphocytes and represent a new model for investigating how lymphoid differentiation may be impaired in the disease state and regulated in the normal state.

Type
dissertation
Published
1983-02-10
Cited by
2,224
References
14

Keywords

Molecular biology, Biology, Humanities, Philosophy

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